A research study has actually discovered that fatty plaques in the arteries that supply the heart, brain, and legs with blood rapidly build up in people between the ages of 40 and 50 years.
A brand-new research study that appears in the Journal of the American College of Cardiology has actually found that atherosclerosis— the process of fatty plaques developing in the arteries that cause the heart and the leading reason for coronary heart disease— quickly increases in individuals between the ages of 40 and 50.
The researchers carried out the research study using imaging methods, showing that ultrasound imaging can be simpler and more effective than other methods.
According to the Centers for Illness Control and Prevention (CDC), atherosclerosis occurs when deposits of cholesterol, along with other compounds, build up in the walls of the arteries, causing these vessels to narrow.
This constricting can decrease the flow of blood or, at its most severe, totally block it. When this occurs in the arteries that provide blood to the heart, it is called coronary cardiovascular disease The symptoms can include chest discomfort, weak point, lightheadedness, pain in the arms, and shortness of breath.
Eventually, this can lead to the weakening of a person’s heart muscle, which can lead to a heart attack
Risk aspects for coronary heart disease consist of cigarette smoking, diabetes, having overweight or weight problems, consuming an unhealthful diet, and having a relative who has heart problem.
In the new study, the researchers examined information that covered 10 years and originated from 4,200 middle-aged men and women who were in health.
They discovered that, contrary to what specialists previously thought, atherosclerosis can happen rapidly between the ages of 40 and 50, even in individuals who otherwise appear healthy.
According to Dr. Borja Ibañez, Clinical Research Study Director of the Centro Nacional de Investigaciones Cardiovasculares (CNIC) in Madrid, Spain: “This study is the very first to examine the development of atherosclerosis at regular intervals. The previous view was